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SSRI may improve metabolic risk factors, hostility: citalopram's benefits include weight loss.


by Wachter, Kerri
Clinical Psychiatry News • April, 2008 • News

BALTIMORE -- For individuals who have high levels of hostility, treatment with citalopram not only reduces hostility but improves the cardiovascular risk factors that make up metabolic syndrome, according to data presented at the annual meeting of the American Psychosomatic Society.

The findings suggest a role for central serotonergic function in the association of hostility with cardiovascular risk factors, said Thomas W. Kamarck, Ph.D., a professor of psychology at the University of Pittsburgh.

"We concluded that it's plausible that individual difference in serotonergic function may in part drive the association between hostility and metabolic risk factors observed in the literature," Dr. Kamarck said.

This analysis draws on data from the larger Stress Treatment and Health Risk (STAHR) project. This placebo-controlled study is aimed at assessing the effect of a selective serotonin re-uptake inhibitor on hostility and cardiovascular measures.

In the study on hostility, 229 healthy adults aged 30-50 years with elevated hostility levels measured using standardized scales were recruited. Exclusion criteria included a history of cardiovascular conditions, a DSM-IV Axis I diagnosis, or use of medication for elevated cholesterol, hypertension, diabetes, or psychiatric disorders.

Participants visited the laboratory five times during a 6-week prerandomization period. During these visits, they were assessed for waist circumference, fasting glucose and insulin, blood pressure, and fasting HDL cholesterol and triglycerides.

The Buss Perry Aggression scale was used to assess hostility. The scale is a self-report measure of hostility that involves four subscales, measuring behavioral (verbal and physical aggression), cognitive (suspiciousness), and affective (angry emotions) aspects of hostility.

In all, 160 participants were then randomized to citalopram (Celexa) or to placebo. During the first 6 weeks of treatment, the subjects randomized to citalopram were titrated up to a maximum dose of 40 mg/day During the next 5 weeks, treatment continued, and the pretreatment assessments were repeated.

The intention-to-treat analysis included 81 participants in the citalopram group and 79 in the placebo group. However, only 72 and 67 participants in the citalopram and placebo groups, respectively, completed the study protocol. The average age was 40 years, and half of the participants were female. The sample was primarily white (84%).

The effect size for reducing hostility was greater for the citalopram group--the effect size was 1.09 for the treatment group versus 0.56 for the placebo group on the Buss Perry Aggression scale, Dr. Kamarck reported.

Treatment with citalopram was associated with greater effect size for waist circumference, glucose, insulin, HDL cholesterol, and triglycerides than for the placebo group.

Hostility previously has been associated with an increased risk for coronary heart disease and all-cause mortality, he said.

A recent meta-analysis showed that hostility is associated with the group of metabolic risk factors for cardiovascular disease, including waist-hip ratio, glucose and insulin resistance, lipid ratios, and triglycerides, that are cumulatively known as metabolic syndrome (Health Psychol. 2006;25:493-500).

Dr. Kamarck and his colleagues next adjusted their models for changes in hostility scores over the study period. They found that changes in hostility levels did not account for changes in metabolic risk factors.

Treatment with citalopram also was associated with a small but significant loss of weight. So the researchers also adjusted their models to account for changes in body mass index.

After that adjustment, only the effect of citalopram on HDL remained significant, he said.

"The serotonergic effects on hostility and metabolic risks are not entirely overlapping. The latter effects may be largely mediated by weight regulation processes, at least among a nondiabetic sample," Dr. Kamarck said.

The results suggest that augmentation of central serotonergic function may benefit an individual with psychosocial risk factors for cardiovascular disease.

Dr. Kamarck said in an interview that he had no conflicts of interest.

BY KERRI WACHTER

Senior Writer


COPYRIGHT 2008 International Medical News Group Reproduced with permission of the copyright holder. Further reproduction or distribution is prohibited without permission.
Copyright 2008 Gale, Cengage Learning. All rights reserved. Gale Group is a Thomson Corporation Company.
NOTE: All illustrations and photos have been removed from this article.


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